PHYSIOLOGICAL AND TRANSCRIPTIONAL RESPONSES OF SEED GERMINATION TO MODERATE DROUGHT IN APOCYNUM VENETUM


Aryl hydrocarbon receptor (AHR)-regulated transcriptomic changes in rats sensitive or resistant to major dioxin toxicities

Abstract Background The major toxic effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) appear to result from dysregulation of mRNA levels mediated by the aryl hydrocarbon receptor (AHR).Dioxin-like chemicals alter expression of numerous genes in liver, but it remains unknown which lie in pathways leading to major toxicities such as hepatotoxicit

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